Supplementary Figure 9 from Integrating <i>In Situ</i> Immune Expression Landscape with Gene Expression–Based Subtyping to Characterize Breast Cancer Heterogeneity

Authors: Mustapha Abubakar, Hela Koka, Priscilla Ming Yi Lee et al.

Publication:

Published: Unknown date

Source: Crossref

Back to Search View Original Cite This Article

Abstract

<jats:p><p>Distribution of immune markers by TP53 mutation status among patients with luminal (A) and non-luminal (B) breast cancer in the Hong Kong Breast Cancer Study.</p></jats:p>

Keywords

breast cancer pdistribution immune markers

Supplementary Figure 9 from Integrating &lt;i&gt;In Situ&lt;/i&gt; Immune Expression Landscape with Gene Expression–Based Subtyping to Characterize Breast Cancer Heterogeneity

Abstract

Keywords

Related Articles

Supplementary Figure 9 from Targeting the NAD&lt;sup&gt;+&lt;/sup&gt; Salvage Pathway Blocks Metabolic Recovery and Enhances β-Lapachone Toxicity in NQO1-Expressing Glioblastoma Cells

Supplementary Figure S3 from Pharmacologic Inhibition of PMS2 Induces MMR Deficiency and Response to Immune Checkpoint Blockade

Supplementary Table S2 from HER2 Heterogeneous Breast Cancer Models Reveal Novel Therapeutic Targets and Subclonal Dynamics during Evolution to Resistance to HER2-Targeted Therapies

Supplementary Table S6 from &lt;i&gt;Helicobacter pylori&lt;/i&gt; Infection and Metabolic Dysfunction–Associated Steatotic Liver Disease: A Prospective Study and Meta-Analysis

Data from HER2 Heterogeneous Breast Cancer Models Reveal Novel Therapeutic Targets and Subclonal Dynamics during Evolution to Resistance to HER2-Targeted Therapies

Supplementary Figure 9 from Integrating <i>In Situ</i> Immune Expression Landscape with Gene Expression–Based Subtyping to Characterize Breast Cancer Heterogeneity

Supplementary Figure 9 from Targeting the NAD<sup>+</sup> Salvage Pathway Blocks Metabolic Recovery and Enhances β-Lapachone Toxicity in NQO1-Expressing Glioblastoma Cells

Supplementary Table S6 from <i>Helicobacter pylori</i> Infection and Metabolic Dysfunction–Associated Steatotic Liver Disease: A Prospective Study and Meta-Analysis